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Showing results 1 to 13 of 13

Issue DateTitle / Author(s) / CitationFileAltmetrics
2001-02
Inhibition of epidermal growth factor-induced cell transformation and activator protein 1 activation by [6]-gingerol

Bode, Ann M.; Ma, Wei-Ya; Surh, Young-Joon; Dong, Zigang

Cancer Research, Vol.61 No.3, pp.850-853

2007-09
Myricetin is a novel natural inhibitor of neoplastic cell transformation and MEK1
DOI
2010-11
Breaking the NF-kB and STAT3 Alliance Inhibits Inflammation and Pancreatic Tumorigenesis

Surh, Young-Joon; Bode, Ann M.; Dong, Zigang

Cancer Prevention Research, Vol.3 No.11, pp.1379-1381

DOI
2013-04
Tumor suppressor p16(INK4a) inhibits cancer cell growth by downregulating eEF1A2 through a direct interaction
DOI
2013-05
Direct Targeting of MEK1/2 and RSK2 by Silybin Induces Cell-Cycle Arrest and Inhibits Melanoma Cell Growth
DOI
2013-08
Tumor suppressor p16 INK4a inhibits cancer cell growth by downregulating eEF1A2 through a direct interaction (vol 125, pg 1744, 2013)
DOI
2013-08
Tumor suppressor p16 INK4a inhibits cancer cell growth by downregulating eEF1A2 through a direct interaction (vol 125, pg 1744, 2013)
DOI
2013-12
Esculetin Suppresses Proliferation of Human Colon Cancer Cells by Directly Targeting beta-Catenin
DOI
2014-03
[6]-Shogaol inhibits growth and induces apoptosis of non-small cell lung cancer cells by directly regulating Akt1/2
DOI
2015-10
Aschantin targeting on the kinase domain of mammalian target of rapamycin suppresses epidermal growth factor-induced neoplastic cell transformation
DOI
2015-11
Genetic ablation of caspase-7 promotes solar-simulated light-induced mouse skin carcinogenesis: the involvement of keratin-17
DOI
2016-10
15-Deoxy-Delta(12,14)-prostaglandin J(2) stabilizes hypoxia inducible factor-1 alpha through induction of heme oxygenase-1 and direct modification ofprolyl-4-hydroxylase 2
DOI
2017-10
hYSK1 promotes cancer cell proliferation and migration through negative regulation of p16INK4a under hypoxic conditions

Lee, Mee-Hyun; Dong, Zigang; Surh, Young-Joon; Choi, Bu Young

Oncotarget, Vol.8 No.51, pp.89072-89085

DOI
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