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Study on neurotoxic mechanisms of C-terminal fragments of amyloid precursor protein through the regulation of GSK-3β transcription in neuronal cells : 신경세포에서 아밀로이드 전구 단백질의 C단 대사물들의 GSK-3β 유전자 발현 조절을 통한 신경독성기전 연구

DC Field Value Language
dc.contributor.advisor서유헌-
dc.contributor.author김은미-
dc.date.accessioned2009-11-30T04:53:58Z-
dc.date.available2009-11-30T04:53:58Z-
dc.date.copyright2004.-
dc.date.issued2004-
dc.identifier.urihttp://dcollection.snu.ac.kr:80/jsp/common/DcLoOrgPer.jsp?sItemId=000000054687-
dc.identifier.urihttps://hdl.handle.net/10371/17172-
dc.descriptionThesis (doctoral)--서울대학교 대학원 :의학과 약리학 전공,2004.en
dc.format.extentviii, 83 leavesen
dc.language.isoen-
dc.publisher서울대학교 대학원en
dc.subjectC-terminal fragment of amyloid precursor proteinen
dc.subject아밀로이드 전구단백질의 C 단 단백질en
dc.subjectAlzherimer`s diseaseen
dc.subject알츠하이머병en
dc.subjectGlycogen synthase kinase 3betaen
dc.subjectGlycogen synthase kinase 3ßen
dc.subjectFe65en
dc.subjectCP2/LSF/LBP1 전사요소en
dc.subjectCP2/LSF/LBP1 transcription factoren
dc.titleStudy on neurotoxic mechanisms of C-terminal fragments of amyloid precursor protein through the regulation of GSK-3β transcription in neuronal cellsen
dc.title.alternative신경세포에서 아밀로이드 전구 단백질의 C단 대사물들의 GSK-3β 유전자 발현 조절을 통한 신경독성기전 연구en
dc.typeThesisen
dc.contributor.department의학과 약리학 전공-
dc.description.degreeDoctoren
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