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Genetic susceptibility in tuberculosis

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dc.contributor.authorYim, Jae-Joon-
dc.contributor.authorSelvaraj, Paramasivam-
dc.date.accessioned2012-05-29T07:52:56Z-
dc.date.available2012-05-29T07:52:56Z-
dc.date.issued2010-02-
dc.identifier.citationRESPIROLOGY; Vol.15 2; 241-256ko_KR
dc.identifier.issn1323-7799-
dc.identifier.urihttps://hdl.handle.net/10371/76568-
dc.description.abstractThe importance of host genetic factors in determining susceptibility to tuberculosis (TB) has been studied extensively using various methods, such as case-control, candidate gene and genome-wide linkage studies. Several important candidate genes like human leucocyte antigen/alleles and non-human leucocyte antigen genes, such as cytokines and their receptors, chemokines and their receptors, pattern recognition receptors (including toll-like receptors, mannose binding lectin and the dendritic cell-specific intercellular adhesion molecule-3 grabbing nonintegrin), solute carrier family 11A member 1 (formerly known as natural resistance-associated macrophage protein 1) and purinergic P2X7 receptor gene polymorphisms, have been associated with differential susceptibility to TB in various ethnic populations. This heterogeneity has been explained by host-pathogen and gene-environment interactions and evolutionary selection pressures. Although the achievements of genetics studies might not yet have advanced the prevention and treatment of TB, researchers have begun to widen their scope of investigation to encompass these practical considerations.ko_KR
dc.language.isoenko_KR
dc.publisherWILEY-BLACKWELL PUBLISHING, INCko_KR
dc.subjectcytokineko_KR
dc.subjecttuberculosisko_KR
dc.subjectgene polymorphismko_KR
dc.subjectsolute carrier family 11A member 1ko_KR
dc.subjecthuman leucocyte antigenko_KR
dc.titleGenetic susceptibility in tuberculosisko_KR
dc.typeArticleko_KR
dc.contributor.AlternativeAuthor임재준-
dc.identifier.doi10.1111/j.1440-1843.2009.01690.x-
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