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Rad52/Rad59-dependent recombination as a means to rectify faulty Okazaki fragment processing

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dc.contributor.authorLee, Miju-
dc.contributor.authorLee, Chul-Hwan-
dc.contributor.authorDemin, Annie Albert-
dc.contributor.authorMunashingha, Palinda Ruvan-
dc.contributor.authorAmangyeld, Tamir-
dc.contributor.authorKwon, Buki-
dc.contributor.authorFormosa, Tim-
dc.contributor.authorSeo, Yeon-Soo-
dc.date.accessioned2024-05-14T06:37:21Z-
dc.date.available2024-05-14T06:37:21Z-
dc.date.created2023-05-09-
dc.date.created2023-05-09-
dc.date.issued2014-05-
dc.identifier.citationJournal of Biological Chemistry, Vol.289 No.21, pp.15064-15079-
dc.identifier.issn0021-9258-
dc.identifier.urihttps://hdl.handle.net/10371/201851-
dc.description.abstractBackground: How faulty Okazaki fragments are repaired remains unclear. Results: The DNA annealing activity of Rad52 and sister chromatid cohesion are important in the repair of faulty Okazaki fragments. Conclusion: Rad52/Rad59-mediated sister chromatid recombination is a major means of repairing faulty Okazaki fragments. Significance: The faithful repair of faulty Okazaki fragments is critical for genome stability. © 2014 by The American Society for Biochemistry and Molecular Biology, Inc.-
dc.language영어-
dc.publisherAmerican Society for Biochemistry and Molecular Biology Inc.-
dc.titleRad52/Rad59-dependent recombination as a means to rectify faulty Okazaki fragment processing-
dc.typeArticle-
dc.identifier.doi10.1074/jbc.M114.548388-
dc.citation.journaltitleJournal of Biological Chemistry-
dc.identifier.wosid000337248100058-
dc.identifier.scopusid2-s2.0-84901447597-
dc.citation.endpage15079-
dc.citation.number21-
dc.citation.startpage15064-
dc.citation.volume289-
dc.description.isOpenAccessY-
dc.contributor.affiliatedAuthorLee, Chul-Hwan-
dc.type.docTypeArticle-
dc.description.journalClass1-
dc.subject.keywordPlusREPLICATION PROTEIN-A-
dc.subject.keywordPlusDOUBLE-STRAND BREAKS-
dc.subject.keywordPlusSISTER-CHROMATID COHESION-
dc.subject.keywordPlusYEAST RAD52 PROTEIN-
dc.subject.keywordPlusDNA-DAMAGE RESPONSE-
dc.subject.keywordPlusESSENTIAL IN-VIVO-
dc.subject.keywordPlusSACCHAROMYCES-CEREVISIAE-
dc.subject.keywordPlusHOMOLOGOUS RECOMBINATION-
dc.subject.keywordPlusEND RESECTION-
dc.subject.keywordPlusGENOME STABILITY-
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  • College of Medicine
Research Area Epigenetics, Heterochromatin, Histone Modifications

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