Publications

Detailed Information

Rad52/Rad59-dependent recombination as a means to rectify faulty Okazaki fragment processing

Cited 16 time in Web of Science Cited 16 time in Scopus
Authors

Lee, Miju; Lee, Chul-Hwan; Demin, Annie Albert; Munashingha, Palinda Ruvan; Amangyeld, Tamir; Kwon, Buki; Formosa, Tim; Seo, Yeon-Soo

Issue Date
2014-05
Publisher
American Society for Biochemistry and Molecular Biology Inc.
Citation
Journal of Biological Chemistry, Vol.289 No.21, pp.15064-15079
Abstract
Background: How faulty Okazaki fragments are repaired remains unclear. Results: The DNA annealing activity of Rad52 and sister chromatid cohesion are important in the repair of faulty Okazaki fragments. Conclusion: Rad52/Rad59-mediated sister chromatid recombination is a major means of repairing faulty Okazaki fragments. Significance: The faithful repair of faulty Okazaki fragments is critical for genome stability. © 2014 by The American Society for Biochemistry and Molecular Biology, Inc.
ISSN
0021-9258
URI
https://hdl.handle.net/10371/201851
DOI
https://doi.org/10.1074/jbc.M114.548388
Files in This Item:
There are no files associated with this item.
Appears in Collections:

Related Researcher

  • College of Medicine
Research Area Epigenetics, Heterochromatin, Histone Modifications

Altmetrics

Item View & Download Count

  • mendeley

Items in S-Space are protected by copyright, with all rights reserved, unless otherwise indicated.

Share